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<SlideText><![CDATA[HEART AND ITS NEIGHBORHOOD NEURAL CREST CELLS  migrate from hindbrain region via pharyngeal arches to conotruncal region of heart. ]]></SlideText>
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<SlideText><![CDATA[BLOOD VESSELS OF THE EMBRYO  (at 26 days) ]]></SlideText>
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<SlideText><![CDATA[AORTIC ARCHES SCHEMATIC 1 2 3 6 4 5 7 iseg Ventral aorta Dorsal aorta ]]></SlideText>
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<SlideText><![CDATA[AORTIC ARCHES SCHEMATIC 3 6 4 1 2 5 7 iseg Ventral aorta Dorsal aorta ]]></SlideText>
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<SlideText><![CDATA[6 3 3 4 4 7 iseg 7 iseg AORTIC ARCHES AND DERIVATIVES  Aortic sac Truncus arteriosus 6 ]]></SlideText>
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<SlideText><![CDATA[6 3 3 4 4 7 iseg 7 iseg AORTIC ARCHES AND DERIVATIVES  ]]></SlideText>
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<SlideText><![CDATA[6 3 3 4 4 7 iseg 7 iseg AORTIC ARCHES AND DERIVATIVES  ]]></SlideText>
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<SlideText><![CDATA[LCC RCC RSC BCA LSC DA AORTIC ARCHES AND DERIVATIVES  ]]></SlideText>
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<SlideText><![CDATA[6 3 3 4 4 7 iseg 7 iseg RECURRENT LARYNGEAL NERVES ]]></SlideText>
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<SlideText><![CDATA[LCC RCC RSC BCA LSC DA RECURRENT LARYNGEAL NERVES ]]></SlideText>
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<SlideText><![CDATA[3 6 4 1 2 5 7 iseg Ventral aorta Dorsal aorta DOUBLE AORTIC ARCH ]]></SlideText>
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<SlideText><![CDATA[DOUBLE AORTIC ARCH: “Vascular ring” Causes airway obstruction, stridor in infancy. ]]></SlideText>
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<SlideText><![CDATA[3 6 4 1 2 5 7 iseg Ventral aorta Dorsal aorta RIGHT AORTIC ARCH ]]></SlideText>
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<SlideText><![CDATA[RIGHT AORTIC ARCH: Mirror image branching ]]></SlideText>
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<SlideText><![CDATA[3 6 4 1 2 5 7 iseg Ventral aorta Dorsal aorta ABERRANT RIGHT SUBCLAVIAN ARTERY ]]></SlideText>
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<SlideText><![CDATA[LCC RCC RSC LSC DA ABERRANT RIGHT SUBCLAVIAN ARTERY ]]></SlideText>
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<SlideText><![CDATA[ABERRANT RIGHT SUBCLAVIAN ARTERY Occurs in 0.5% of people. Frequently asymptomatic, it may cause obstructive symptoms. ]]></SlideText>
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<SlideText><![CDATA[COARCTATION OF THE AORTA Notching of ribs on X-ray Blood pressure difference Upper extr >>Lower extr. Associated with: 45X: Turner’s synrome Trisomy 21: Down’s syndrome  70% have bicuspid aortic valve  >90% post (juxta) ductal ]]></SlideText>
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<SlideText><![CDATA[ABDOMINAL ARTERIES ]]></SlideText>
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<SlideText><![CDATA[Umbilical	 Vitelline VENOUS SYSTEM SCHEMATIC Sinus Venosus Cardinal ]]></SlideText>
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<SlideText><![CDATA[© 2005 Elsevier  UMBILICAL AND VITELLINE VEINS- I: Liver, portal vein and ductus venosus.  ]]></SlideText>
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<SlideText><![CDATA[UMBILICAL AND VITELLINE VEINS- II: Liver, portal vein and ductus venosus.  ]]></SlideText>
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<SlideText><![CDATA[Cardinal Subcardinal Supra cardinal Supra-Subcardinal Anastomosis Umbilical	 Vitelline VENOUS SYSTEM SCHEMATIC Sinus Venosus C P A ]]></SlideText>
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<SlideText><![CDATA[THE CARDINAL VEINS AND THE VENAE CAVAE ]]></SlideText>
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<SlideText><![CDATA[THE CARDINAL VEINS AND THE VENAE CAVAE ]]></SlideText>
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<SlideText><![CDATA[SINUS VENOSUS AND THE CORONARY SINUS ]]></SlideText>
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<SlideText><![CDATA[VENOUS (SMOOTH WALLED) PART OF ATRIA ]]></SlideText>
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<SlideText><![CDATA[PERSISTENT LEFT SVC  0.3% of general population. 4 % of patients with Cong. Ht Dis.  Usually drains to Coronary sinus. Usually asymptomatic.  Enlarged coronary sinus is a clue.    ]]></SlideText>
<Notes><![CDATA[]]></Notes>
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<SlideText><![CDATA[LYMPHATIC SYSTEM ]]></SlideText>
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<Title><![CDATA[Slide 30]]></Title>
<SlideText><![CDATA[FETAL CIRCULATION ]]></SlideText>
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<Title><![CDATA[Slide 31]]></Title>
<SlideText><![CDATA[POSTNATAL CIRCULATION ]]></SlideText>
<Notes><![CDATA[]]></Notes>
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<Title><![CDATA[Slide 32]]></Title>
<SlideText><![CDATA[PATENT DUCTUS ARTERIOSUS Prostaglandins:  Keep the ductus Patent Indomethacin: Closes the ductus.  Physiologic closure: Normally 82% by 48hrs, 100% by 4 days. Anatomic closure: 12 weeks.  Patent Ductus :	-prematurity. 		-neonatal hypoxic states. 		-maternal Rubella infection ]]></SlideText>
<Notes><![CDATA[]]></Notes>
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<Title><![CDATA[Slide 33]]></Title>
<SlideText><![CDATA[CELL LINEAGES IN HEART DEVELOPMENT FHF = First Heart Field; SHF = Second Heart Field; CNC: Cardiac Neural Crest Ref: Srivastava, D.  Making or Breaking the Heart: From Lineage Determination to Morphogenesis.Cell 126, Sep. 22, 2006 p1037-1048. ]]></SlideText>
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<Title><![CDATA[Slide 34]]></Title>
<SlideText><![CDATA[MOLECULAR PATHWAYS IN HEART DEVELOPMENT Ref: Srivastava, D.  Making or Breaking the Heart: From Lineage Determination to Morphogenesis.Cell 126, Sep. 22, 2006 p1037-1048. ]]></SlideText>
<Notes><![CDATA[]]></Notes>
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<Title><![CDATA[Slide 35]]></Title>
<SlideText><![CDATA[Genetic Mutation Syndrome Name Cardiac Disease            Nonsyndromic Nonsyndromic Nonsyndromic            NKX2-5 — Atrial septal defect, ventricular septal defect, electrical conduction defect            GATA4 — Atrial septal defect, ventricular septal defect            MYH6 — Atrial septal defect            NOTCH1 — Aortic valve disease            Syndromic Syndromic Syndromic            TBX5 Holt-Oram Atrial septal defect, ventricular septal defect, electrical conduction defect            TBX1 DiGeorge Cardiac outflow tract defect            TFAP2β Char Patent ductus arteriosus            JAG1 Alagille Pulmonary artery stenosis, tetralogy of Fallot            PTPN11 Noonan Pulmonary valve stenosis            Elastin William Supravalvar aortic stenosis            Fibrillin Marfan Aortic aneurysm            Genetic Mutations in Congenital Heart Disease  Ref: Srivastava, D.  Making or Breaking the Heart: From Lineage Determination to Morphogenesis.Cell 126, Sep. 22, 2006 p1037-1048. ]]></SlideText>
<Notes><![CDATA[]]></Notes>
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<Title><![CDATA[Slide 36]]></Title>
<SlideText><![CDATA[MOVIE! ]]></SlideText>
<Notes><![CDATA[]]></Notes>
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